- Oxford Library of Psychology
- Oxford Library of Psychology
- About the Editors
- Introduction to The Oxford Handbook of Cognitive Neuroscience: Cognitive Neuroscience: Where Are We Now?
- Salience, State, and Expression: The Influence of Specific Aspects of Emotion on Attention and Perception
- Emotion: Generation or Construction?
- The Neuroscience of Emotion Regulation: Basic Mechanisms and Their Role in Development, Aging, and Psychopathology
- The Impact of Emotion on Cognition
- Genetics and Emotion
- Visceromotor Sensation and Control
- Development of Emotion and Social Reasoning in Adolescence
- Perception of Nonverbal Cues
- Face Recognition
- The Cognitive and Neural Basis of Impression Formation
- Theory of Mind: How Brains Think about Thoughts
- The Pleasures and Pains of Social Interactions: A Social Cognitive Neuroscience Perspective
- The Neural Underpinnings of the Experience of Empathy: Lessons for Psychopathy
- Mirror Neurons and the Perception–Action Link
- The Early Development of the Brain Bases for Social Cognition
- Conflict Monitoring and Cognitive Control
- Hierarchical Cognitive Control and the Functional Organization of the Frontal Cortex
- Decision Neuroscience
- Expectancies and Beliefs: Insights from Cognitive Neuroscience
- Cognitive Neuroscience of Numerical Cognition
- Psychopharmacology of Cognition
- Attention Deficit/Hyperactivity Disorder (ADHD)
- Post-Traumatic Stress Disorder (PTSD)
- Generalized Anxiety Disorder and Social Phobia Considered from a Cognitive Neuroscience Perspective
- The Neurobiology of Obsessive-Compulsive Disorder
- Epilogue to The Oxford Handbook of Cognitive Neuroscience Cognitive Neuroscience: Where Are We Going?
Abstract and Keywords
Generalized anxiety disorder (GAD) and social phobia (SP) are two of the major anxiety disorders identified by DSM-IV. They are highly comorbid and overlap in symptoms yet present with distinct features (e.g., worry in GAD and fear of embarrassment in SP). Given the overlap in symptoms and suggestions that conditioning-based accounts may explain all anxiety disorders, this chapter first evaluates whether hyperconditioning or hypersensitivity to threat is found in both or either disorder. On the basis of the current literature, this cannot currently be concluded in the case of GAD but may be the case for SP, as least for social-threat stimuli. The chapter then examines potential neurocognitive functions that might be aberrant in specific ways in the two disorders. Much remains to be discovered before any adequate cognitive neuroscience account of GAD can be considered. However, one form of neurocognitive function that appears markedly aberrant in SP relates to self-referential processing.
Karina S. Blair, Department of Health and Human Services, National Institute of Mental Health, National Institutes of Health, Bethesda, MD
R.J.R. Blair, Department of Health and Human Services, National Institute of Mental Health, National Institutes of Health, Bethesda, MD
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