Abstract and Keywords
Communication of social-affective intent through vocal modulation (prosody) has received increasing recent attention from cognitive neuroscientists. Clinically, dysprosodia is a cardinal feature of schizophrenia and may also be present in bipolar disorder. This chapter summarizes the state of knowledge regarding schizophrenia and bipolar dysprosodia, examining how it is measured and the neural mechanisms that underlie its disturbance. The authors argue that in schizophrenia, rather than reflecting generalized emotional dysfunction, affective prosody deficits are better explained from an information theoretical perspective of impaired audio-linguistic signal processing (ALSP) beginning with basic impairments to simple pitch perception that, along with higher-order cognitive impairments, generate dysprosodia. The ALSP model engenders specific theoretical and clinical implications, which the chapter also details. Finally, the chapter outlines the limitations of the ALSP model and current approaches that examine dysprosdia in single individuals, advocating that future research must study prosody within a communication and linguistic perspective that examines interpersonal communications.
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